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As long as that cell survives or gives rise to new cells, the FeLV proviral DNA persists and leaves behind a blueprint for infectious virus. Studies using polymerase chain reaction (PCR) for detecting proviral DNA copies of FeLV indicate that most cats are unlikely to eliminate this infection.1,2.

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Although the etiology and pathophysiology of INOCA and HFpEF are complex and not fully understood, there is no doubt that the underlying cause of both conditions is endothelial dysfunction (ED) which further promotes the development of left ventricular diastolic dysfunction (LVDD).

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